Publications of the Human and Animal Physiology Group
Researchers of Human and Animal Physiology publish on a regular basis in reputed peer reviewed international scientific journals.
Our publications
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Ndufs4 knockout mice with isolated complex I deficiency engage a futile adaptive brain response
Biochimica et Biophysica Acta - Proteins and Proteomics (2025), Volume: 1873, Issue: 1 - ISSN 1570-9639 -
The impact of forearm immobilization and acipimox administration on muscle amino acid metabolism and insulin sensitivity in healthy, young volunteers
American Journal of Physiology. Endocrinology and Metabolism (2024), Volume: 326, Issue: 3 - ISSN 0193-1849 - p. E277-E289. -
Performance of TMRM and Mitotrackers in mitochondrial morphofunctional analysis of primary human skin fibroblasts
Biochimica et Biophysica Acta - Bioenergetics (2024), Volume: 1865, Issue: 2 - ISSN 0005-2728 -
Relationship between Volitional and Non-Volitional Quadriceps Muscle Endurance in Patients with Chronic Obstructive Pulmonary Disease
Diagnostics (2024), Volume: 14, Issue: 2 - ISSN 2075-4418 -
The Hypoxia Response Pathway : A Potential Intervention Target in Parkinson's Disease?
Movement Disorders (2024), Volume: 39, Issue: 2 - ISSN 0885-3185 - p. 273-293. -
Early-Life Exposure to Dietary Large Phospholipid-Coated Lipid Droplets Improves Markers of Metabolic and Immune Function in Adipose Tissue Later in Life in a Mouse Model
Molecular Nutrition and Food Research (2024), Volume: 68, Issue: 1 - ISSN 1613-4125 -
Nasogastric bolus administration of a protein-rich drink augments insulinaemia and aminoacidaemia but not whole-body protein turnover or muscle protein synthesis versus oral administration
Clinical science (2024), Volume: 138, Issue: 1 - ISSN 0143-5221 - p. 43-60. -
Translatability of mouse muscle-aging for humans : the role of sex
GeroScience (2024), Volume: 46, Issue: 3 - ISSN 2509-2715 - p. 3341-3360. -
TLCD4 as Potential Transcriptomic Biomarker of Cold Exposure
Biomolecules (2024), Volume: 14, Issue: 8 - ISSN 2218-273X -
Maternal riboflavin deficiency causes embryonic defects by activating ER stress-induced hepatocyte apoptosis pathway
Free Radical Biology and Medicine (2024), Volume: 224 - ISSN 0891-5849 - p. 418-435.